Gerald C Hsu

The author describes his hypothesis on the probable partial self-recovery of some insulin regeneration capability of pancreatic
beta cells on a Type 2 diabetes (T2D) patient via his collected data of both postprandial plasma glucose (PPG) and fasting plasma
glucose (FPG) during the period of 2014 to 2019. As a T2D patient for 25 years, he took various diabetes medications to control his
elevated glucose levels since 1998. For the last 20 years, he has suffered many T2D complications, including five cardiac episodes
and renal complications, except for having a stroke. Starting from 2013, he reduced the dosages of his three prescribed diabetes
medications. In August 2015, he discontinued taking his last remaining medication, the classical metformin HCL. In other words, his
body has been free of any chemical compound from medications or insulin injections for four years. Since then, he has completely
relied on a stringent lifestyle management program to control his diabetes conditions. As a result, his T2D has been under control
(HbA1C ~6.5%) since 2016. He has kept nearly two million data of his own medical conditions and lifestyle details. He also developed
sophisticated computer software by using artificial intelligence to analyze, process, and manage this massive health data.
The author observed improvement in his diabetes conditions after following a stringent lifestyle management since 2014. From
examining his own glucose data in 2018 including the existing vulnerable conditions of his “damaged” beta cells due to his high
carbs/sugar intake, he hypothesized that beta cells are still able to “repair” themselves to a certain degree. This “dual-phenomena”
can be observed with his higher open and close PPG values and his ultra-high PPG values when he violated his own strict control
rules of diet and exercise during the same period of pancreatic beta cells partial recovery.


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